>New York State Legislative Action Alert > >In January 1999, Senate Bill No. S-982 was introduced by State Senators >Seward and Skelos. If passed, this bill will require the use of the >chicken pox vaccine for children in New York. You will no longer have the >right to object to chicken pox vaccination because it will become one >more on the list of vaccines required to get into school. > >WHAT YOU CAN DO > >1---Besides clipping, signing and mailing this letter to your state >senator, make a phone call to his/her local district office. Say that you >oppose Bill No. S-982. In your own words, tell them why you believe this >legislation should not be passed. > >2---Send an email to your state senator. Address the email to „[LAST >NAME]@senate.state.ny.us¾. For example, if your senator is Frank Padavan, >then address the email to „padavan@senate.state.ny.us¾. Be sure to >include your complete name and address on your e-mail. If you do not know >who your state senator is, call your public library or log onto the >internet at „http://www.senate.ny.us¾ > >3---Send the same email to Senators Seward (seward @senate.state.ny.us) >and Skelos (skelos@senate.state.ny.us), the co-sponsors of Bill No. >S-982, telling them you oppose this bill. > >4---Make copies of this action alert and pass it out to as many people as >possible. Ask them to send letters in their own words, with the same >message, and to identify themselves as parents or grandparents if they >are. > > >==================================== > > > >The Honorable ____________________ >New York State Senate >Albany, NY 12247 > >Dear Senator _____________, > >As a parent and concerned citizen, I oppose the passage of Senate Bill >No. S-982, introduced by State Senators Seward and Skelos, that would >make the chicken pox vaccine mandatory for children. My vote in the next >senatorial election will depend solely on your position on this issue. > >Safety Considerations >Long-term studies have yet to be conducted to prove the safety and >effectiveness of chicken pox vaccine over a child¼s lifetime. Since it >was discovered in 1971 that biological substances entering directly into >the bloodstream could become part of human genetic structure, scientists >have been concerned about the long-term effects of viral DNA, from >live-virus vaccines, being incorporated into human genes. Aside from >these foreign viral particles, there are short term dangers posed by >vaccines, according Money magazine,12/96. Vaccines contain highly >antigenic (toxic) proteins, as well as extremely poisonous preservatives, >adjuvants, neutralizers, carrying agents and extracting agents, all >injected directly into the bloodstreams of two, four, and six month old >infantsãwhose immune systems are not fully developedãbypassing their >immune system, especially the liver, whose purpose it is to filter such >poisons. Since the government undertook to insure vaccine manufacturers >over a decade ago, compensation payments for vaccine injuries to children >have averaged about $2-3 million per week. > >Chicken Pox Vaccine Not Worth The Risk >„Many primary care physicians either do not recommend this vaccine, or >suggest that children be immunized only if they have not developed >chicken pox by 12 years of age¾ (Jrnl Amer Med Assc, 11/97). > >The American Academy of Pediatrics states in its brochure on chickenpox >(1996), „Most children who are otherwise healthy and get chickenpox won¼t >have any complications from the disease.¾ Of the approximately 4 million >cases of chickenpox in the United States each year, the death rate is >only .0025%, with a hospitalization rate of .23%, making it one of the >most benign diseases in existence. Adults are almost 10 times more likely >than children under 14 to need hospitalization from chickenpox and more >than 20 times more likely to die from the disease.¾ Yet the vaccine is >not mandated for adults. > >The primary justification made by the manufacturer, Merck & Co., and the >American Academy of Pediatrics, is the economic consideration of parental >work loss, or the inconvenience of a child missing a soccer game or >birthday party, rather than on any pressing public health issues. > >Public health officials insist that protecting the immunocompromised >population is an important consideration for the mandate of the vaccine. >However, this group represents one-tenth of one percent of total >chickenpox cases. Moreover, studies are only now underway to assess the >feasibility of vaccinating this population safely. Therefore, the >necessity to vaccinate all children at $30 per shot, with a vaccine of >questionable efficacy, is without justification. > >Deaths Are Not From Chicken Pox >The only medical justification for this vaccine has been the claimed >mortalities from the disease. However, chicken pox is not a fatal >disease, but rather a very common, benign inflammatory condition. >Fatalitiesãas rare as they areãactually result from inappropriate >treatment; usually the kinds of aggressive medical interventions as >described in Figure 1. For each of the three deaths described, to >understand why the children¼s medical conditions deteriorated following >every drug application, one need only read how the famed clinician, Henry >Bieler, M.D., described chicken pox: > >Chicken pox arises from the elimination of toxic fat or fatty acids >through the hair fat glands. The chemical burn from the purging of waste >products though the skin causes the characteristic blister of this >disease. This occurs when the liver is congested and cannot perform its >eliminative function, metabolic waste matter (toxins) is thrown into the >bloodstream. These toxins in the blood must be discharged, so nature uses >vicarious avenues of elimination, or „substitutes¾. When these bile >poisons (from the liver) in the blood come out through the skin, we get >skin conditions manifested by rashes, boils, acne, etc. Or they come out >through the mucous membranes (inside skin) manifesting as various >catarrhs, like chicken pox. The disease is determined by the chemistry of >the poison being eliminated. Thus, the skin is „substituting¾ for the >liver, or a vicarious elimination is occurring through the skin. > >Food And Drugs Are Contraindicated >During the more acute and involved forms of toxemia, such as measles, >chicken pox, fever, or flu, the liver is much too busy neutralizing toxic >wastes to be bothered with digestion of food. Therefore, to facilitate >the elimination of this waste, fasting on distilled water is essential in >such cases. This accounts for the lack of digestive juices produced, and >the loss of appetite that accompanies these illnesses. > >After cells have been damaged by the toxic wastes, it is important for >bacteriaãacting as scavengersãto attack and devour the weakened, injured >and dead cells. Otherwise, these dead cells would become accumulated >toxic waste themselves. Therefore, antibiotics and other bactericides >must not be administered. The so called „bad¾ bacterial strains die out >on their own anyway, once their food (toxic waste) is used up. But until >that point, they play an important role in the process that converts >waste for eventual elimination. > >The class of drugs that doctors use to treat catarrhs like chicken pox >are called antipyretics. Among antipyretics, aspirin tops the list of >favorites. Aspirin is a phenol (carbolic acid) derivative, with all the >chemical qualities of phenol, but without the deadly effect of carbolic >acid. Aspirin, like phenol, deadens the nerve endings, thereby masking >pain. Aspirin also diminishes a fever by partially blocking the thyroid >and the adrenal glands. But the phenol derivatives interfere with the >proper function of the liver and damage liver cells. The use of aspirin, >then, is an attempt to drive out one devil (disease toxins) by admitting >another devil! > >The Importance Of Fever >Fever in a child is a frightening symptom to the mother. Just what is the >function of fever? Is it a harmful process, something to suppress and >worry about? Or is it the body's attempt to burn up a poison, thereby >helping to dispose of it more quickly? > >In the diseases of childhood, fever begins in the liver. In a very >strong, robust child, with properly functioning endocrine glands, the >toxin is often completely consumed in the liver. The child does not feel >sick or have pain; it just has a fever and if the liver area is carefully >palpated, it can be noted that there is an elevation of temperature over >that organ. In fact, if the temperature under the tongue is 105 degrees, >the internal temperature of the liver may be as high as 110 degrees. But >if the liver is unable to oxidize completely the poisons of disease so >that some leak through into the blood stream, then, under the action of >the endocrine glands, the poisons seek vicarious outlets via the mucous >membranes. This may be through the upper respiratory tract, in the form >of flu, sinusitis, pharyngitis, tonsillitis and possibly even pneumonia, >which is a complicated kind of bronchitis. All through this process, the >whole power of the liver is diverted into neutralizing the toxic wastes >of disease, as evidenced by the fever. > >The liver is much too busy to be bothered with the task of the digestion >of food. Great strain can be taken off that organ if no food is given. >Not only does fasting lower the temperature, relieve the distress and >facilitate elimination, but it also lessens the strain on the liver and >prevents serious complications, such as middle ear disease, mastoiditis >and meningitis. Left alone, a fever will not exceed 106 degrees. Only >about 4 percent of children experience fever-related convulsions, with no >serious aftereffects. > >A fast (on distilled water, or at least diluted fruit or vegetable >juices) should be continued for twenty four hours after the temperature >has returned to normal. A good rule to remember is that the bowel can be >cleared of toxins (by physic or enemas) in twenty four hours; the blood >in three days; the liver in five days, providing no food is eaten. >Shingles („adult chicken pox¾), another eliminative crisis through the >mucous membranes occuring in adults, requires at least an week-long fast >to completely clear up. > >It appears then, that fever, dreaded because misunderstood, is really >nature's attempt to help. It is discomforting, but never does harm; never >is attended with serious aftereffects and never should be suppressed with >anti-inflammatory drugs or fed with food. I have seen many a case of flu >pushed into a pneumonia because some anxious grandmother insisted upon >something "to give the child strength¾, such as chicken broth or a thin >starchy gruel, both liquids, of course, but protein and starchãjust what >the liver cannot handle at this point. > >From, „Food Is Your Best Medicine¾, 1966 > >Chicken Pox Doesn¼t Kill; Doctors Kill >It¼s now plain to see why the children described by the CDC in Fig.1 had >died. They were given numerous antibiotics, antipyretic and antipruritic >medications, other fever suppressants, and probably food. The CDC admits >that children don¼t die from chicken pox per se, but rather >„complications¾ from chicken pox. But what they don¼t say is that these >complications are all derived from acute blood toxemia established by the >treatments of allopathic physicians: > >Not surprisingly, the CDC lists as the most common: pneumonia and >secondary bacterial infections (caused by the antibiotics). Other >complications, according to the CDC, include „encephalitis¾ (inflamed >brain tissue mostly from the antipyretics), „hemorrhagic complications¾ >(such as intestinal bleeding, are the most common symptoms of aspirinãan >anticoagulant, or „blood thinner¾), hepatitis (congested and inflamed >liver caused by the antipyretics), arthritis (decalcification of bone for >the calcium needed to neutralize acidic blood mostly caused by the >aspirin), and Reye¼s syndrome (most commonly associated with giving >aspirin to children that have chicken pox or influenza). Prescribing >acetaminophen (Tylenol, etc.) in large doses are also toxic to the liver >and kidneys, because they also check the vital actions of the body to >discharge waste from the blood. > >To promote the vaccine, the CDC proclaims that, „Varicella (chicken pox) >is the leading cause of vaccine-preventable deaths in children in the >United States.¾ But while the deaths are certainly preventable, it has >nothing to do with the vaccine! > >Published by Coalition For Informed Choice (CFIC), PO Box 230426, >Holliswood, NY 11423, 718-479-2939 (ph+fax), > >Endorsed By: > >Parent: ____________________________________ > >Address: ___________________________________ > > > >=====================BOX==================== >Morbidity and Mortality Weekly Report >May 15, 1998 / Vol. 47/No. 18 > >Varicella-Related Deaths Among Children >Texas and Iowa notified CDC of three fatal cases of varicella >(chickenpox) that occurred in children during 1997 > >Case 1 > >On February 28, 1997, a previously healthy, unvaccinated 21-month-old boy >developed a typical varicella rash. He had no reported exposure to >varicella. On March 1, he was taken to a local emergency department (ED) >with a high fever and was started on oral acetaminophen and >diphenhydramine. On March 3, his primary-care physician prescribed oral >acyclovir. On March 4, his mother noted a new petechial-like rash. The >next morning, his primary-care physician noted lethargy, a purpuric rash, >and poor perfusion. He was transferred to a local ED. Fluid resuscitation >and intravenous ceftriaxone were initiated, but the child continued to >deteriorate rapidly, requiring intubation, mechanical ventilation, and >inotropic support with dopamine. Blood cultures were negative for >bacterial pathogens. Laboratory tests indicated disseminated >intravascular coagulation and severe dehydration. Approximately 1.5 hours >after arrival at the ED, he was transported to a tertiary-care center. >Within 10 minutes of arrival, he suffered cardiac arrest and died. The >death was attributed to varicella with hemorrhagic complications. > >Case 2 > >On December 21, 1997, a 5-year-old unvaccinated boy with a history of >asthma was taken to a local ED with a fever of 104.5 F (40.3 C) and a >typical varicella rash in multiple stages of healing. The child was >treated with antipyretic and antipruritic medications and discharged. > >That evening, the boy developed mild dyspnea and was treated at home for >a presumed asthma attack with metered-dose inhalers and one dose of oral >prednisone. He returned to the ED on December 22 with shortness of breath >and a 4-hour history of abdominal and leg pain. On presentation to the >ED, one of the patient¼s siblings had active varicella and another had >recently recovered from varicella. Physical examination revealed numerous >chickenpox lesions, one of which appeared infected. He was tachypneic, >and his extremities were mottled consistent with peripheral septic >emboli. Chest and abdominal radiographs revealed a right pleural >effusion, pneumonia, and mild ileus. Thoracostomy produced pleural fluid >containing gram-positive cocci, confirmed 8 hours later to be group A >Streptococcus (GAS). A peripheral blood sample revealed gram-positive >cocci. He was admitted to the hospital and treated with intravenous >ceftriaxone, nafcillin, and acyclovir. > >After admission, his breathing became labored and his extremities >increasingly mottled. He rapidly developed hypotension, obtundation, and >bradycardia. Despite efforts at cardiopulmonary resuscitation, the child >died 5 hours after arriving at the ED. A post-mortem examination >attributed the death to GAS septicemia, pneumonia, and pleural effusion, >complicating varicella infection. > >Case 3 > >On December 14, 1996, a previously healthy, unvaccinated 23-month-old boy >developed fever and a typical varicella rash. Approximately 1-2 weeks >earlier, his unvaccinated 4-year-old sibling had contracted varicella. He >was taken to his physician on December 17 because of persistent fever and >cellulitis of the left foot, and he was hospitalized on December 19 for >failure to improve on an unspecified outpatient antibiotic regimen. >Because his condition deteriorated despite intravenous methicillin and >ceftriaxone, he was transferred to a regional hospital on December 21. >Sepsis, possible viral meningoencephalitis, and mild pleural effusion >were diagnosed. A cerebrospinal fluid examination revealed lymphocytic >pleocytosis, and blood and urine cultures grew penicillin-resistant >Staphylococcus aureus. Antibiotics were changed to nafcillin and >gentamycin, and intravenous acyclovir was added on December 23. On >December 24, the child developed an aortic insufficiency murmur, and an >echocardiogram revealed a 9x9 mm vegetation on the aortic valve, >consistent with bacterial endocarditis. Serial echocardiograms displayed >growth of the vegetation and development of a pericardial effusion. He >was transferred to a cardiac surgery center on December 26. While >awaiting surgery, he developed refractive heart failure secondary to >staphylococcal endocarditis. He became incoherent, probably secondary to >a major embolic neurologic event, and died on January 8, 1997. > >====================BOX====================== >CAPTION: >In 1997, 3 deaths reported by two states did not occur from chicken pox, >but rather from the unnecessary drugs they used to treat it. >==============================================